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Troponin Through The Looking-glass
Myofilament Ca2+ Sensitization Causes Susceptibility to Cardiac Arrhythmia in          Mice
Myosin Rod Hypophosphorylation and CB Kinetics in Papillary Muscles from a TnC-A8V KI Mouse Model.
Late Onset Sporadic Dilated Cardiomyopathy Caused by a Cardiac Troponin T          Mutation
Enhanced troponin I binding explains the functional changes produced by the hypertrophic cardiomyopathy mutation A8V of cardiac troponin C.
Fluorescent Protein-Based Ca2+ Sensor Reveals Global, Divalent Cation-Dependent Conformational Changes in Cardiac Troponin C
Heart Disease and Depression in Females Estrous Cycle Influence over Senstivity to Ketamine’s Antidepressant                 Effects
Characterization of Cardiac Troponin C FRET Constructs to Analyze Structural Changes Induced by Divalent Cation                 Binding
Amide Hydrogens Reveal A Temperature-dependent Structural Transition That Enhances Site-ii Ca2+ -binding Affinity In A C-domain Mutant Of Cardiac Troponin C
Amide hydrogens reveal a temperature-dependent structural transition that enhances site-II Ca(2+)-binding affinity in a C-domain mutant of cardiac troponin C.
Genotype-specific pathogenic effects in human dilated cardiomyopathy.
Constitutive phosphorylation of cardiac myosin regulatory light chain in vivo.
Long term ablation of protein kinase A (PKA)-mediated cardiac troponin I phosphorylation leads to excitation-contraction uncoupling and diastolic dysfunction in a knock-in mouse model of hypertrophic cardiomyopathy.
Hypertrophic Cardiomyopathy Cardiac Troponin C Mutations Differentially Affect Slow Skeletal And Cardiac Muscle Regulation
In Vivo Analysis of Troponin C Knock-In (A8V) Mice