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Method of treating multiple sclerosis with anti-K6 antibody
Mutant polypeptides of fibroblast growth factor 1
Mutant polypeptides of fibroblast growth factor 1
Mutants of human fibroblast growth factor having increased stability and/or mitogenic potency
Mutants of human fibroblast growth factor having increased stability and/or mitogenic potency
Mutant polypeptides of fibroblast growth factor 1
Mutants of human fibroblast growth factor having increased stability and/or mitogenic potency
Mutants of human fibroblast growth factor having increased stability and/or mitogenic potency
Small animal restraining harness or jacket
Fibroblast growth factor mutants having improved functional half-life and methods of their use
Fibroblast growth factor mutants having improved functional half-life and methods of their use
Fibroblast growth factor mutants having improved functional half-life and methods of their use
9783587
Engineered human acidic fibroblast growth factors and associated methods
Alternative Folding Nuclei Definitions Facilitate the Evolution of a Symmetric Protein Fold from a Smaller Peptide Motif
Activation Profiles of Human Kallikrein-Related Peptidases by Proteases of the          Thrombostasis Axis
Accelerated healing in NONcNZO10/LtJ type 2 diabetic mice by FGF 1
Folding Nucleus Structure Persists in Thermally-Aggregated FGF-1
Evolution and design of protein structure by folding nucleus symmetric expansion
Expression and Function of the Kallikrein-Related Peptidase 6 in the Human Melanoma          Microenvironment
Fine-sampled Photographic Quantitation of Dermal Wound Healing Senescence in Aged BALB/cByJ Mice and Therapeutic Intervention with FGF-1
Experimental support for the foldability-function tradeoff hypothesis
Functional Role of Kallikrein 6 in Regulating Immune Cell Survival
Mathematical Model for the Determination of Mouse Excisional Wound Healing Parameters from Photographic Data
Kallikrein 6 Signals through PAR1 and PAR2 to Promote Neuron Injury and Exacerbate Glutamate Neurotoxicity
Kallikrein 6 is a Novel Molecular Trigger of Reactive Astrogliosis
Isomannide-based peptidomimetics as inhibitors for human tissue kallikreins 5 and 7.
Kallikrein-related Peptidase 6
Kallikreins are Associated with Secondary Progressive Multiple Sclerosis and Promote          Neurodegeneration
Investigating the Dynamics and Polyanion Binding Sites of Fibroblast Growth Factor-1 Using Hydrogen-Deuterium Exchange Mass Spectrometry
Molecular Modeling of Substrate Binding in Wild-type and Mutant Corynebacteria 2,5-diketo-D-gluconate Reductases
Myths and Verities in Protein Folding Theories
Mutagenesis of the Crystal Contact of Acidic Fibroblast Growth Factor
Mutation Choice to Eliminate Buried Free Cysteines in Protein Therapeutics
Pharmacokinetic properties of 2nd-generation fibroblast growth factor-1 mutants for therapeutic application.
Pharmacokinetic Properties of 2(nd)-Generation Fibroblast Growth Factor-1 Mutants for          Therapeutic Application
Completed KLK Activome Profile
Crystal Structure and Biochemical Characterization of Human Kallikrein 6 Reveals a Trypsin-like Kallikrein is Expressed in the Central Nervous System
Emergence of Symmetric Protein Architecture from a Simple Peptide Motif
Engineering a Cysteine-Free Form of Human Fibroblast Growth Factor-1 for "2nd Generation" Therapeutic Application
Engineering a Cysteine-Free Form of Human Fibroblast Growth Factor-1 for "Second Generation" Therapeutic Application.
S(1)' and S(2)' Subsite Specificities of Human Plasma Kallikrein and Tissue Kallikrein          1 for the Hydrolysis of Peptides Derived from the Bradykinin Domain of Human          Kininogen
S116R Phosphorylation Site Mutation in Human FGF-1 Differentially Affects Mitogenic and Glucose Lowering Activities
Simplified protein design biased for prebiotic amino acids yields a foldable, halophilic protein.
Protease-Activated Receptor Dependent and Independent Signaling by Kallikreins 1 and 6          in CNS Neuron and Astroglial Cell Lines
Substrate Specificity of Human Kallikreins 1 and 6 Determined by Phage Display
SYMMETRIC PROTEIN ARCHITECTURE IN PROTEIN DESIGN: TOP-DOWN SYMMETRIC DECONSTRUCTION
Simplified Protein Design Biased for Pre-Biotic Amino Acids Yields a Foldable, Halophilic Protein
Prebiotic Protein Design supports a Halophile Origin of Foldable Proteins
Experimental Support for the Evolution of Symmetric Protein Architecture from a Simple          Peptide Motif

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