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14-3-3τ promotes surface expression of Cav2.2 (α1B) Ca2+ channels.
Long term ablation of protein kinase A (PKA)-mediated cardiac troponin I phosphorylation leads to excitation-contraction uncoupling and diastolic dysfunction in a knock-in mouse model of hypertrophic cardiomyopathy.
Intracellular regions of the Eag potassium channel play a critical role in generation of voltage-dependent currents.
Cdc45 protein-single-stranded DNA interaction is important for stalling the helicase during replication stress.
Allosteric Transmission along a Loosely Structured Backbone Allows a Cardiac Troponin C Mutant to Function with Only One Ca Ion.
replication initiation protein Sld2 regulates helicase assembly.
Positive Amplification Mechanism Involving a Kinase and Replication Initiation Factor Helps Assemble the Replication Fork Helicase.
DNA Interactions Probed by Hydrogen-Deuterium Exchange (HDX) Fourier Transform Ion Cyclotron Resonance Mass Spectrometry Confirm External Binding Sites on the Minichromosomal Maintenance (MCM) Helicase.
Dpb11 protein helps control assembly of the Cdc45·Mcm2-7·GINS replication fork helicase.
DNA sequences proximal to human mitochondrial DNA deletion breakpoints prevalent in human disease form G-quadruplexes, a class of DNA structures inefficiently unwound by the mitochondrial replicative Twinkle helicase.
Constitutive phosphorylation of cardiac myosin regulatory light chain in vivo.
Transmembrane Domain Mediates Tetramerization of α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid (AMPA) Receptors.
Replication Initiation Protein Sld3/Treslin Orchestrates the Assembly of the Replication Fork Helicase during S Phase.
Stoichiometric relationship among clock proteins determines robustness of circadian rhythms.