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14-3-3τ promotes surface expression of Cav2.2 (α1B) Ca2+ channels.
Allosteric Transmission along a Loosely Structured Backbone Allows a Cardiac Troponin C Mutant to Function with Only One Ca Ion.
Cdc45 protein-single-stranded DNA interaction is important for stalling the helicase during replication stress.
Constitutive phosphorylation of cardiac myosin regulatory light chain in vivo.
DNA Interactions Probed by Hydrogen-Deuterium Exchange (HDX) Fourier Transform Ion Cyclotron Resonance Mass Spectrometry Confirm External Binding Sites on the Minichromosomal Maintenance (MCM) Helicase.
DNA sequences proximal to human mitochondrial DNA deletion breakpoints prevalent in human disease form G-quadruplexes, a class of DNA structures inefficiently unwound by the mitochondrial replicative Twinkle helicase.
Dpb11 protein helps control assembly of the Cdc45·Mcm2-7·GINS replication fork helicase.
Intracellular regions of the Eag potassium channel play a critical role in generation of voltage-dependent currents.
Long term ablation of protein kinase A (PKA)-mediated cardiac troponin I phosphorylation leads to excitation-contraction uncoupling and diastolic dysfunction in a knock-in mouse model of hypertrophic cardiomyopathy.
Positive Amplification Mechanism Involving a Kinase and Replication Initiation Factor Helps Assemble the Replication Fork Helicase.
replication initiation protein Sld2 regulates helicase assembly.
Replication Initiation Protein Sld3/Treslin Orchestrates the Assembly of the Replication Fork Helicase during S Phase.
Stoichiometric relationship among clock proteins determines robustness of circadian rhythms.
Transmembrane Domain Mediates Tetramerization of α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid (AMPA) Receptors.